Muscle spasm may be connected with many disease states of the spine and internal organs. In some cases, localized muscle spasm may be a protective physiological mechanism limiting the mobility of the involved spinal segments. But then constricted muscles become a secondary source of pain, which starts a vicious circle of pain-muscle spasm-pain “that promotes the formation of myofascial pain syndrome MFBS.
The leading pathogenetic mechanisms MFBS include local vasomotor dysfunction and reflex tonic changes in the whole muscle or its limited area.
Reflex syndrome includes acute lumbago with disease development and sciatica with subacute or chronic course. When it occurs, these pains are characterized by flattening the lumbar lordosis. This is the first stage of neurological complications. From compression syndromes most common are radiculopathy, which accounted for 40% of all extra vertebral syndromes. In connection with the physiological characteristics of the structure of the spine the lumbar section is most commonly affected. Any damage to the vertebral-motor segment at the lumbar spine in the body begin sanogenetic reaction to limit the movement in the affected segment, which leads to a change in the motor stereotype, which is formed due to the close interaction between pyramidal and extrapyramidal systems.
Next stage or stage of discogenic sciatica is based on a disc prolapse and increased penetration of the disc tissue in the epidural space, where the spinal roots are. Emerging with radicular symptoms correspond to the level of the affected vertebral segment. Most often it affects the roots L5 and S1.
The following stage of neurological disorders caused by continuing compression in the spine hernia and passing with it is radicular artery. It can develop quickly catastrophically “paralytic sciatica”, characterized by peripheral paresis or paralysis of the extensor muscles of the foot. In such cases, the appearance of motor disturbances is accompanied by the disappearance of pain.
The fourth stage of neurological manifestations is caused by circulatory disturbance of the spinal cord due to damage of root-spinal because of tax arteries. Most disturbed blood flow is in the Wojciech Adamkiewicz artery and additional Deprozh-Guttheron artery. It develops chronic discirculatory myelopathy, usually at the lumbar spinal cord. The patient in case of lesion appeared in Wojciech Adamkiewicz artery syndrome, has intermittent claudication of the spinal cord. Any damage to the Deprozh-Gutterona artery develops a syndrome of intermittent claudication of horse’s tail.
Worse is case when vertebrogen lumbar radicular syndrome is compression of cauda equina, the so-called caudal syndrome. Most often it is caused by fallen midline disc herniation, which compresses all the roots at the affected segment.
Degenerative-dystrophic diseases of the spine (that is to say, is often in combination with prolapse or disc herniation) leads to the development of segmental spinal and radicular canals stenosis. The most frequent cause of spinal stenosis is a combination of progressive degenerative processes in the discs, joints and ligaments of the spine with the antecedent of relatively low capacity of the channel as a result of congenital or constitutional peculiarities of the structure of the vertebral bodies.
To learn more about sciatica and sciatic nerve pain in particular – please visit this site with helpful sciatica-related info.
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